Cytokines & Infection -The best immune response is not always the strongest. Surviving an infection requires an immune response strong enough to kill the pathogen, but not so strong that the collateral damage kills the infected person too.
We're working to understand how that balance is achieved. We are particularly interested in cytokines, and the interaction between coincident, opposing cytokine signals. How does cytokine cross-talk impose immune balance, or dictate the outcome of co-infection? Can we manipulate key cytokine signals for therapeutic gain? Follow us on Twitter @PeronaLab
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